Please use this identifier to cite or link to this item: https://hdl.handle.net/1959.11/4002
Title: Nutritional manipulation of necrotic enteritis outbreak in broilers
Contributor(s): Kocher, Andreas (author)
Publication Date: 2003
Handle Link: https://hdl.handle.net/1959.11/4002
Abstract: A significant threat to the broiler industry is the sporadic outbreak of necrotic enteritis (NE). Damage to the intestinal mucosa through coccidial :infection ('Eimeria' spp.) or a change in the normal intestinal microflora predisposes birds to the proliferation of 'Clostridium perfringens' (CP) resulting in NE. At present in Australia the occurrence of NE in poultry is still controlled by adding antibacterial growth promotants (AGP) to feed. However, the ban of AGP in many European countries and recent trends to reduce and ban throughout the world the inclusion of AGP in all animal feed has forced the poultry industry worldwide to look for different means by which to control the disease. The choice of feed ingredient can significantly alter the intestinal microflora and predispose or protect birds from the rapid proliferation of potential pathogens like CP More recently, the focus has shifted towards the inclusion of alternative feed supplements such as feed enzymes, prebiotic, probiotic and organic acids, which not only improve broiler performance but also provide considerable protection against NE.
Publication Type: Conference Publication
Conference Details: RAAN 2003: Recent Advances in Animal Nutrition in Australia, Armidale, Australia, 13th - 16th July, 2003
Source of Publication: Recent Advances in Animal Nutrition in Australia, v.14, p. 111-116
Publisher: University of New England
Place of Publication: Armidale, Australia
ISSN: 0819-4823
Fields of Research (FoR) 2008: 070204 Animal Nutrition
Socio-Economic Objective (SEO) 2008: 830309 Poultry
Peer Reviewed: Yes
HERDC Category Description: E1 Refereed Scholarly Conference Publication
Publisher/associated links: http://trove.nla.gov.au/work/33151111?selectedversion=NBD41064497
Appears in Collections:Conference Publication

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